By Benjamin Caballero, Richard J. Wurtman (auth.), Richard J. Wurtman M.D., Eva Ritter-Walker (eds.)
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Extra resources for Dietary Phenylalanine and Brain Function
109-140. , and Haber, M. (1976a). Use of aspartame in phenylketonuric heterozygous adults. J. Toxicol. Environ. Health 2:453-457. F. (1976b). Results of loading doses of 3. Effect of Aspartame on Plasma Phe Levels 39 aspartame by two phenylketonuric (PKU) children compared with two normal children. J. Toxico!. Environ. Health 2:459-469. Matalon, R (1986). Aspartame intake in carriers for phenylketonuria. In Proceedings of the international aspartame workshop, Session IV, Hyperphenylalaninemia, Chapter 3.
113:1851-1860. L. (1987a). Repeated ingestion of aspartame-sweetened beverage by normal adult subjects: effect on plasma amino acid concentration. Metabolism (in press). E. (1987b). Plasma amino acid concentrations in normal adults administered aspartame in capsules or solution: lack of bioequivalence. Metabolism 36: 507-512. L. (1987c). Plasma amino acid concentrations in normal adults ingesting aspartame and monosodium L-glutamate as part of a soup-beverage meal. Metabolism 36: 1073-1079. E. (1987d).
25 ILmoVdl in normal subjects and PKU heterozygotes, respectively. When the number of normal subjects and heterozygous subjects was increased, similar results were noted (Stegink et al. 1981b). From the shape of the curves describing uptake and clearance of phenylalanine from plasma, it is evident that PKU heterozygotes clear phenylalanine less rapidly. 33 ILmoVdl, respectively. Based upon the observation that the plasma concentration-time curve of heterozygotes administered APM at 34 mglkg body weight was similar to that of normal subjects administered APM at 50 mglkg body weight, we concluded that heterozygotes metabolized the phenylalanine portion of aspartame approximately one-half as well as normal subjects.